CaMKII inhibition due to TRIC-B loss-of-function dysregulates SMAD signaling in osteogenesis imperfecta-Reference-Cited by-同舟云学术

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CaMKII inhibition due to TRIC-B loss-of-function dysregulates SMAD signaling in osteogenesis imperfecta

Published:2023-06 Issue: Volume:120 Page:43-59
ISSN:0945-053X
Container-title:Matrix Biology
language:en
Short-container-title:Matrix Biology

Author:

Besio Roberta^ORCID,Contento Barbara M.^ORCID,Garibaldi Nadia,Filibian Marta^ORCID,Sonntag Stephan,Shmerling Doron,Tonelli Francesca,Biggiogera Marco,Brini Marisa^ORCID,Salmaso Andrea,Jovanovic Milena^ORCID,Marini Joan C.,Rossi Antonio,Forlino Antonella^ORCID

Publisher

Elsevier BV

Subject

Molecular Biology

Reference53 articles.

1. TRIC channels are essential for Ca2+ handling in intracellular stores;Yazawa;Nature,2007

2. Essential role of the TRIC-B channel in Ca2+ handling of alveolar epithelial cells and in perinatal lung maturation;Yamazaki;Development,2009

3. TRIC-B: an under-explored druggable ion channel;Hu;Nat. Rev. Drug Discov.,2019

4. Enhanced activity of multiple TRIC-B channels: an endoplasmic reticulum/sarcoplasmic reticulum mechanism to boost counterion currents;O'Brien;J. Physiol.,2019

5. The journey of Ca2+ through the cell-pulsing through the network of ER membrane contact sites;Cremer;J. Cell Sci.,2020

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Update on the Genetics of Osteogenesis Imperfecta;Calcified Tissue International;2024-08-11

2. Absence of TRIC-B from type XIV Osteogenesis Imperfecta osteoblasts alters cell adhesion and mitochondrial function – A multi-omics study;Matrix Biology;2023-08

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