p-TAK1 acts as a switch between myoblast proliferation phase and differentiation phase in mdx mice via regulating HO-1 expression
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Published:2022-10
Issue:
Volume:933
Page:175277
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ISSN:0014-2999
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Container-title:European Journal of Pharmacology
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language:en
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Short-container-title:European Journal of Pharmacology
Author:
Fan Shusheng,
Huang Xiaofei,
Tong Haowei,
Hong Huitao,
Lai Zhulan,
Hu Wanting,
Liu Xiaoyun,
Zhang Luyong,
Jiang ZhenzhouORCID,
Yu Qinwei
Reference31 articles.
1. Assessing functional performance in the mdx mouse model;Aartsma-Rus;JoVE,2014
2. Absence of dystrophin disrupts skeletal muscle signaling: roles of Ca2+, reactive oxygen species, and nitric oxide in the development of muscular dystrophy;Allen;Physiol. Rev.,2016
3. The "usual suspects": genes for inflammation, fibrosis, regeneration, and muscle strength modify Duchenne muscular dystrophy;Bello;J. Clin. Med.,2019
4. Function and genetics of dystrophin and dystrophin-related proteins in muscle;Blake;Physiol. Rev.,2002
5. Diagnosis and management of Duchenne muscular dystrophy, part 1: diagnosis, and neuromuscular, rehabilitation, endocrine, and gastrointestinal and nutritional management;Birnkrant;Lancet Neurol.,2018
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