Angiotensin II limits NO production by upregulating arginase through a p38 MAPK–ATF-2 pathway-Reference-Cited by-同舟云学术

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Angiotensin II limits NO production by upregulating arginase through a p38 MAPK–ATF-2 pathway

Published:2015-01 Issue: Volume:746 Page:106-114
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Shatanawi Alia,Lemtalsi Tahira,Yao Lin,Patel Chintan,Caldwell Ruth B.,Caldwell R.William

Funder

National Institutes of Health

University of Jordan Research Deanship

2012 L׳Oréal-UNESCO For Women in Science Pan-Arab Regional Fellowship

2014 L׳Oréal-UNESCO For Women in Science International Fellowship

Publisher

Elsevier BV

Subject

Pharmacology

Reference32 articles.

1. Measurement of nitric oxide in biological models;Archer;Faseb. J.,1993

2. Arginase pathway in human endothelial cells in pathophysiological conditions;Bachetti;J. Mol. Cell Cardiol.,2004

3. Treatment with the arginase inhibitor N(omega)-hydroxy-nor-l-arginine improves vascular function and lowers blood pressure in adult spontaneously hypertensive rat;Bagnost;J. Hypertens.,2008

4. Arginase reciprocally regulates nitric oxide synthase activity and contributes to endothelial dysfunction in aging blood vessels;Berkowitz;Circulation,2003

5. Overexpression of arginase in the aged mouse penis impairs erectile function and decreases eNOS activity: influence of in vivo gene therapy of anti-arginase;Bivalacqua;Am. J. Physiol. Heart Circ. Physiol.,2007

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