Intracellular zinc status influences cisplatin-induced endothelial permeability through modulation of PKCα, NF-κB and ICAM-1 expression

Author:

Bodiga Vijaya Lakshmi,Inapurapu Santhi Priya,Vemuri Praveen Kumar,Kudle Madhukar Rao,Bodiga Sreedhar

Funder

UGC-Startup Grant

Publisher

Elsevier BV

Subject

Pharmacology

Reference71 articles.

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2. Zinc decreases C-reactive protein, lipid peroxidation, and inflammatory cytokines in elderly subjects: a potential implication of zinc as an atheroprotective agent;Bao;Am. J. Clin. Nutr.,2010

3. Nitric-oxide-mediated zinc release contributes to hypoxic regulation of pulmonary vascular tone;Bernal;Circ. Res.,2008

4. Physiological roles of zinc in the plasma membrane of mammalian cells;Bettger;J. Nutr. Biochem.,1993

5. Silencing of PKC-alpha, TRPC1 or NF-kappaB expression attenuates cisplatin-induced ICAM-1 expression and endothelial dysfunction;Bodiga;Biochem. Pharm.,2015

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