Tunicamycin inhibits Toll-like receptor-activated inflammation in RAW264.7 cells by suppression of NF-κB and c-Jun activity via a mechanism that is independent of ER-stress and N-glycosylation-Reference-Cited by-同舟云学术

Tunicamycin inhibits Toll-like receptor-activated inflammation in RAW264.7 cells by suppression of NF-κB and c-Jun activity via a mechanism that is independent of ER-stress and N-glycosylation

Published:2013-12 Issue:1-3 Volume:721 Page:294-300
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Kim Song-Yi,Hwang Ji-Sun,Han Inn-Oc

Funder

National Research Foundation of Korea (NRF)

Korea Government (MEST)

Inha University

Publisher

Elsevier BV

Subject

Pharmacology

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