The TMEM16A blockers benzbromarone and MONNA cause intracellular Ca2+-release in mouse bronchial smooth muscle cells

Author:

Dwivedi Ritu,Drumm Bernard T.,Alkawadri Tuleen,Martin S. Lorraine,Sergeant Gerard P.,Hollywood Mark A.,Thornbury Keith D.ORCID

Funder

European Union

Publisher

Elsevier BV

Subject

Pharmacology

Reference64 articles.

1. ER-localized bestrophin 1 activates Ca2+-dependent ion channels TMEM16A and SK4 possibly by acting as a counterion channel;Barro-Soria;Pflügers Archiv,2010

2. New selective inhibitors of calcium-activated chloride channels - T16A(inh) -A01, CaCC(inh) -A01 and MONNA - what do they inhibit?;Boedtkjer;Br. J. Pharmacol.,2015

3. Pharmacological characterization of TMEM16A currents;Bradley;Channels,2014

4. Modulation of rabbit airway smooth muscle responsiveness by respiratory epithelium. Involvement of an inhibitory metabolite of arachidonic acid;Butler;Am. Rev. Respir. Dis.,1987

5. TMEM16A, a membrane protein associated with calcium-dependent chloride channel activity;Caputo;Science,2008

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