Matrine blocks AGEs- induced HCSMCs phenotypic conversion via suppressing Dll4-Notch pathway-Reference-Cited by-同舟云学术

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Matrine blocks AGEs- induced HCSMCs phenotypic conversion via suppressing Dll4-Notch pathway

Published:2018-09 Issue: Volume:835 Page:126-131
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Liu Zhongwei,Wang Yuan,Zhu Haitao,Qiu Chuan,Guan Gongchang,Wang Junkui,Guo Yan

Funder

Special Financial Grant from the China Postdoctoral Science Foundation

China Postdoctoral Science Foundation

National Scientific Foundation of China

Sailing Foundation

Publisher

Elsevier BV

Subject

Pharmacology

Reference42 articles.

1. A glycation angle to look into the diabetic vasculopathy: cause and cure;Ahmad;Curr. Vasc. Pharmacol.,2017

2. Notch transcriptional control of vascular smooth muscle regulatory gene expression and function;Basu;J. Biol. Chem.,2013

3. Vascular smooth muscle cells in atherosclerosis;Bennett;Circ. Res.,2016

4. HES1, a target of Notch signaling, is elevated in canine osteosarcoma, but reduced in the most aggressive tumors;Dailey;BMC Vet. Res.,2013

5. Advanced glycation end-products: implications for diabetic and non-diabetic nephropathies;Daroux;Diabetes Metab.,2010

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1. Disproportionately higher cardiovascular disease risk and incidence with high fructose corn syrup sweetened beverage intake among black young adults–the CARDIA study;Nutrition Journal;2024-07-29

2. The potential therapeutic value of the natural plant compounds matrine and oxymatrine in cardiovascular diseases;Frontiers in Cardiovascular Medicine;2024-07-08

3. Low‐intensity pulsed ultrasound combined with ST36 modulate gastric smooth muscle contractile marker expression via RhoA/Rock and MALAT1/miR‐449a/DLL1 signaling in diabetic rats;Neurogastroenterology & Motility;2024-06-14

4. Research Advancements in Pharmacological Activities and Mechanisms of Matrine;Pharmacognosy Magazine;2023-10-27

5. AGEs induce high expression of Dll4 via endoplasmic reticulum stress PERK signaling-mediated internal ribosomal entry site mechanism in macrophages;Heliyon;2023-10

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