AGEs induce high expression of Dll4 via endoplasmic reticulum stress PERK signaling-mediated internal ribosomal entry site mechanism in macrophages-Reference-Cited by-同舟云学术

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AGEs induce high expression of Dll4 via endoplasmic reticulum stress PERK signaling-mediated internal ribosomal entry site mechanism in macrophages

Published:2023-10 Issue:10 Volume:9 Page:e21170
ISSN:2405-8440
Container-title:Heliyon
language:en
Short-container-title:Heliyon

Author:

Ma Yanpeng,Zheng Shixiang,Wang Xiqiang,Zhu Ling,Wang Junkui,Pan Shuo,Zhang Yong,Liu Zhongwei^ORCID

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference21 articles.

1. Advanced glycation end products induce atherosclerosis via RAGE/TLR4 signaling mediated-M1 macrophage polarization-dependent vascular smooth muscle cell phenotypic conversion;Xing;Oxid. Med. Cell. Longev.,2022

2. Macrovascular complications of type 2 diabetes mellitus;Viigimaa;Curr. Vasc. Pharmacol.,2020

3. Advanced glycation end products (AGE) and diabetes: cause, effect, or both?;Vlassara;Curr. Diabetes Rep.,2014

4. Advanced glycation endproducts and plaque instability: a link beyond diabetes;Paneni;Eur. Heart J.,2014

5. Macrophage phenotype and function in different stages of atherosclerosis;Tabas;Circ. Res.,2016

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Comprehensive research on the properties of advanced glycation end products in food and biological samples and their harmful role in inducing metabolic diseases;Comprehensive Reviews in Food Science and Food Safety;2024-08-13

2. Diphthamide – a conserved modification of eEF2 with clinical relevance;Trends in Molecular Medicine;2023-12

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