Glycogen synthase kinase-3β negatively regulates TGF-β1 and Angiotensin II-mediated cellular activity through interaction with Smad3-Reference-Cited by-同舟云学术

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Glycogen synthase kinase-3β negatively regulates TGF-β1 and Angiotensin II-mediated cellular activity through interaction with Smad3

Published:2010-10 Issue:1-3 Volume:644 Page:17-23
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Hua Fang,Zhou Junlan,Liu Jinwen,Zhu Chuanjiang,Cui Bing,Lin Heng,Liu Yuying,Jin Wen,Yang Hongzhen,Hu Zhuowei

Funder

National Major Basic Research Program of China

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology

Reference25 articles.

1. Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo;Antos;Proc. Natl. Acad. Sci. U. S. A.,2002

2. Stable expression of a human HSP70 gene in a rat myogenic cell line confers protection against endotoxin;Chi;Am. J. Physiol. Cell Physiol.,1996

3. Akt interacts directly with Smad3 to regulate the sensitivity to TGF-beta induced apoptosis;Conery;Nat. Cell Biol.,2004

4. TRB3: a tribbles homolog that inhibits Akt/PKB activation by insulin in liver;Du;Science,2003

5. Glycogen synthase kinase 3: a key regulator of cellular fate;Forde;Cell Mol. Life Sci.,2007

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