GSK-3 at the heart of cardiometabolic diseases: Isoform-specific targeting is critical to therapeutic benefit-Reference-Cited by-同舟云学术

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GSK-3 at the heart of cardiometabolic diseases: Isoform-specific targeting is critical to therapeutic benefit

Published:2023-08 Issue:6 Volume:1869 Page:166724
ISSN:0925-4439
Container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
language:en
Short-container-title:Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease

Author:

Umbarkar Prachi,Ruiz Ramirez Sulivette Y.,Toro Cora Angelica,Tousif Sultan,Lal Hind

Funder

National Heart, Lung, and Blood Institute

Publisher

Elsevier BV

Subject

Molecular Biology,Molecular Medicine

Reference114 articles.

1. Glycogen synthase kinase-3 from rabbit skeletal muscle. Separation from cyclic-AMP-dependent protein kinase and phosphorylase kinase;Embi;Eur. J. Biochem.,1980

2. Molecular cloning and expression of glycogen synthase kinase-3/factor A;Woodgett;EMBO J.,1990

3. Modulation of the glycogen synthase kinase-3 family by tyrosine phosphorylation;Hughes;EMBO J.,1993

4. Mitogen inactivation of glycogen synthase kinase-3 beta in intact cells via serine 9 phosphorylation;Stambolic;Biochem. J.,1994

5. Phosphorylation by p38 MAPK as an alternative pathway for GSK3beta inactivation;Thornton;Science,2008

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