The ability of asymmetric dimethylarginine (ADMA) or monomethylarginine (L-NMMA) to block endothelium-dependent, nitric oxide-mediated relaxation in rat aorta is inversely related to the efficacy of the relaxant stimulus
Author:
Funder
Iraqi Ministry of Higher Education and Scientific Research
Publisher
Elsevier BV
Subject
Pharmacology
Reference22 articles.
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2. Stimulus-specific blockade of nitric oxide-mediated dilatation by asymmetric dimethylarginine (ADMA) and monomethylarginine (L-NMMA) in rat aorta and carotid artery;AL-Zobaidy;Eur. J. Pharmacol.,2011
3. Asymmetric dimethylarginine and critical illness;Brinkmann;Curr. Opin. Clin. Nutr. Metab. Care,2014
4. Elevation of an endogenous inhibitor of nitric oxide synthesis in experimental congestive heart failure;Feng;Cardiovasc. Res.,1998
5. Selective inhibition of basal but not agonist-stimulated activity of nitric oxide in rat aorta by NG-monomethyl-l-arginine;Frew;Br. J. Pharmacol.,1993
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