Stimulus-specific blockade of nitric oxide-mediated dilatation by asymmetric dimethylarginine (ADMA) and monomethylarginine (L-NMMA) in rat aorta and carotid artery
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference38 articles.
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1. Reduction of NO-mediated Relaxing Effects in the Thoracic Aorta in an Experimental Chronic Kidney Disease Mouse Model;Journal of Atherosclerosis and Thrombosis;2015
2. The ability of asymmetric dimethylarginine (ADMA) or monomethylarginine (L-NMMA) to block endothelium-dependent, nitric oxide-mediated relaxation in rat aorta is inversely related to the efficacy of the relaxant stimulus;European Journal of Pharmacology;2014-10
3. Polygonum viviparum L. induces vasorelaxation in the rat thoracic aorta via activation of nitric oxide synthase in endothelial cells;BMC Complementary and Alternative Medicine;2014-05-07
4. The contribution of VEGF signalling to fostamatinib-induced blood pressure elevation;British Journal of Pharmacology;2014-04-11
5. Nitric oxide depletion alters hematopoietic stem cell commitment toward immunogenic dendritic cells;Biochimica et Biophysica Acta (BBA) - General Subjects;2013-03
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