Effect of an oral α2-adrenergic blocker (MK-912) on pancreatic islet function in non-insulin-dependent diabetes mellitus-Reference-Cited by-同舟云学术

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Effect of an oral α2-adrenergic blocker (MK-912) on pancreatic islet function in non-insulin-dependent diabetes mellitus

Published:1991-11 Issue:11 Volume:40 Page:1160-1167
ISSN:0026-0495
Container-title:Metabolism
language:en
Short-container-title:Metabolism

Author:

Ortiz-Alonso F.Javier,Herman William H.,Gertz Barry J.,Williams Vanessa C.,Smith Marla J.,Halter Jeffrey B.

Publisher

Elsevier BV

Subject

Endocrinology,Endocrinology, Diabetes and Metabolism

Reference26 articles.

1. Exaggerated peripheral responsivity to catecholamines contributes to stress-induced hyperglycemia in the obob mouse;Kuhn;Physiol Biochem Behav,1987

2. Adrenergic mechanisms in the hyperglycemia and hyperinsulinemia of diabetic KK mice;Fujimoto;Diabetologia,1981

3. A role for α-adrenergic receptors in abnormal insulin secretion in diabetes mellitus;Robertson;J Clin Invest,1976

4. α-Adrenergic blockade improves glucose-potentiated insulin secretion in non-insulin dependent diabetes mellitus;Broadstone;Diabetes,1987

5. Effects of α-adrenoceptor antagonists on clonidine-induced inhibition of insulin secretion by isolated pancreatic islets;Langer;Br J Pharmacol,1983

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4. MK912☆;Reference Module in Biomedical Sciences;2016

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