Insulin receptor mutation at tyrosines 1162 and 1163 alters both receptor serine phosphorylation and desensitization
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
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2. The stressful condition as a nutritionally dependent adaptive dichotomy;Nutrition;1999-04
3. Insulin induction of protein kinase C alpha expression is independent of insulin receptor Tyr(1162/1163) residues and involves mitogen-activated protein kinase kinase 1 and sustained activation of nuclear p44(MAPK);ENDOCRINOLOGY;1998
4. Insulin Induction of Protein Kinase Cα Expression Is Independent of Insulin Receptor Tyr1162/1163 Residues and Involves Mitogen-Activated Protein Kinase Kinase 1 and Sustained Activation of Nuclear p44MAPK*;Endocrinology;1998-07-01
5. Severe resistance to insulin and insulin-like growth factor-I in cells from a patient with leprechaunism as a result of two mutations in the tyrosine kinase domain of the insulin receptor;Metabolism;1996-12
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