Insulin Induction of Protein Kinase Cα Expression Is Independent of Insulin Receptor Tyr1162/1163 Residues and Involves Mitogen-Activated Protein Kinase Kinase 1 and Sustained Activation of Nuclear p44MAPK*-Reference-Cited by-同舟云学术

Insulin Induction of Protein Kinase Cα Expression Is Independent of Insulin Receptor Tyr1162/1163 Residues and Involves Mitogen-Activated Protein Kinase Kinase 1 and Sustained Activation of Nuclear p44MAPK*

Published:1998-07-01 Issue:7 Volume:139 Page:3133-3142
ISSN:0013-7227
Container-title:Endocrinology
language:en
Short-container-title:

Author:

Antoine Pierre Jacques¹,Bertrand France¹,Auclair Martine¹,Magré Jocelyne¹,Capeau Jacqueline¹,Cherqui Gisèle¹

Affiliation:

1. INSERM U-402, Institut Federatif de Recherche 65, Laboratoire de Biologie Cellulaire, Faculté de Médecine Saint-Antoine, 75571 Paris Cedex 12, France

Publisher

The Endocrine Society

Subject

Endocrinology

Link

http://academic.oup.com/endo/article-pdf/139/7/3133/10348066/endo3133.pdf

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4. Insulin receptor tyrosine residues 1162 and 1163 control insulin stimulation of myristoyl-diacylglycerol generation and subsequent activation of glucose transport.;Cherqui;J Biol Chem,1990

5. A further comparison of insulin- and phorbol ester-stimulated glucose transport in adipocytes.;Cherqui;Mol Cell Endocrinol,1989

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