Lipopolysaccharide potentiates endothelin-1-induced proliferation of pulmonary arterial smooth muscle cells by upregulating TRPC channels-Reference-Cited by-同舟云学术

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Lipopolysaccharide potentiates endothelin-1-induced proliferation of pulmonary arterial smooth muscle cells by upregulating TRPC channels

Published:2016-08 Issue: Volume:82 Page:20-27
ISSN:0753-3322
Container-title:Biomedicine & Pharmacotherapy
language:en
Short-container-title:Biomedicine & Pharmacotherapy

Author:

Jiang Hong-Ni,Zeng Bo,Chen Gui-Lan,Lai Bin,Lu Shao-Hua,Qu Jie-Ming

Funder

Shanghai Leading Talent Projects

Shanghai Education Committee

Natural Science Foundation of Shanghai

Shanghai Leading Academic Discipline Project

National Natural Science Foundation of China

Department of Science & Technology of Sichuan Province

Luzhou City and Luzhou Medical College

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference40 articles.

1. Surviving Sepsis Campaign Guidelines Committee including the Pediatric, Surviving sepsis campaign: international guidelines for management of severe sepsis and septic shock: 2012;Dellinger;Crit. Care Med.,2013

2. Vascular biology in sepsis: pathophysiological and therapeutic significance of vascular dysfunction;Matsuda;J. Smooth Muscle Res.,2007

3. The role of the endothelium in modulating vascular control in sepsis and related conditions;Wort;Br. Med. Bull.,1999

4. Voltage-independent calcium channels mediate lipopolysaccharide-induced hyporeactivity to endothelin-1 in the rat aorta;El-Awady;Am. J. Physiol. Heart Circ. Physiol.,2009

5. Ventricular dysfunction and dilation in severe sepsis and septic shock: relation to endothelial function and mortality;Furian;J. Crit. Care,2012

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3. Calcineurin Is a Universal Regulator of Vessel Function—Focus on Vascular Smooth Muscle Cells;Cells;2023-09-13

4. Deletion of classical transient receptor potential 1, 3 and 6 alters pulmonary vasoconstriction in chronic hypoxia-induced pulmonary hypertension in mice;Frontiers in Physiology;2022-12-07

5. TRPC channels blockade abolishes endotoxemic cardiac dysfunction by hampering intracellular inflammation and Ca2+ leakage;Nature Communications;2022-12-02

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