Mitochondrial dysfunction, AMPK activation and peroxisomal metabolism: A coherent scenario for non-canonical 3-methylglutaconic acidurias-Reference-Cited by-同舟云学术

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Mitochondrial dysfunction, AMPK activation and peroxisomal metabolism: A coherent scenario for non-canonical 3-methylglutaconic acidurias

Published:2020-01 Issue: Volume:168 Page:53-82
ISSN:0300-9084
Container-title:Biochimie
language:en
Short-container-title:Biochimie

Author:

Vamecq Joseph,Papegay Bérengère,Nuyens Vincent,Boogaerts Jean,Leo Oberdan,Kruys Véronique

Funder

French Institut National de la Recherche Scientifique

Inserm

CHU of Lille

University of Nord de France

Belgian Université Libre de Bruxelles

CHU Marie Curie of Charleroi

Publisher

Elsevier BV

Subject

General Medicine,Biochemistry

Reference482 articles.

1. Induction of oxidative stress by the metabolites accumulating in 3-methylglutaconic aciduria in cerebral cortex of young rats;Leipnitz;Life Sci.,2008

2. Cerebrospinal fluid organic acids in biotinidase deficiency;Duran;J. Inherit. Metab. Dis.,1993

3. NMR spectroscopic studies on the late onset form of 3-methylglutaconic aciduria type I and other defects in leucine metabolism;Engelke;NMR Biomed.,2006

4. Experimental evidence that 3-methylglutaric acid disturbs mitochondrial function and induced oxidative stress in rat brain synaptosomes: new converging mechanisms;Colín-González;Neurochem. Res.,2016

5. Redox homeostasis is compromised in vivo by the metabolites accumulating in 3-hydroxy-3-methylglutaryl-CoA lyase deficiency in rat cerebral cortex and liver;da Rosa;Free Radic. Res.,2013

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4. Myelodysplastic syndrome in a patient with Barth syndrome (3‐methylglutaconic aciduria type II);Pediatric Blood & Cancer;2022-10-02

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