CCDC134 ameliorated experimental autoimmune encephalomyelitis by suppressing Th1 and Th17 cells-Reference-Cited by-同舟云学术

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CCDC134 ameliorated experimental autoimmune encephalomyelitis by suppressing Th1 and Th17 cells

Published:2018-07 Issue: Volume:71 Page:158-168
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Xia Peng,Gong Xiaoting,Xiao Lin,Wang Yida,Zhang Tianzhuo,Liao Qinyuan,Mo Xiaoning,Qiu Xiaoyan,Huang Jing

Funder

National Natural Sciences Foundation of China

Beijing Natural Sciences Foundation

National Basic Research Program of China

National Science and Technology Major Projects of New Drugs

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference39 articles.

1. SOCS1 mimetics and antagonists: a complementary approach to positive and negative regulation of immune function;Ahmed;Front. Immunol.,2015

2. Cytokine and chemokine alterations in tissue, CSF, and plasma in early presymptomatic phase of experimental allergic encephalomyelitis (EAE), in a rat model of multiple sclerosis;Borjini;J. Neuroinflammation,2016

3. Chemokines and chemokine receptors in multiple sclerosis;Cheng;Mediators Inflammation,2014

4. Effect of targeted disruption of STAT4 and STAT6 on the induction of experimental autoimmune encephalomyelitis;Chitnis;J. Clin. Invest.,2001

5. Therapeutic targeting of STAT pathways in CNS autoimmune diseases;Egwuagu;Jak-Stat,2013

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5. Total Flavonoids of Astragalus Inhibit Activated CD4+ T Cells and Regulate Differentiation of Th17/Th1/Treg Cells in Experimental Autoimmune Encephalomyelitis Mice by JAK/STAT and NFκB Signaling Pathways;The American Journal of Chinese Medicine;2023-01

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