A central role for protein kinase C overactivity in diabetic glomerulosclerosis: implications for prevention with antioxidants, fish oil, and ACE inhibitors

Author:

McCarty M.F.

Publisher

Elsevier BV

Subject

General Medicine

Reference166 articles.

1. Protein kinase C is activated in glomeruli from streptozotocin diabetic rats: possible mediation by glucose;Craven;J Clin Invest,1989

2. Increase in diacylglycerol mass in isolated glomeruli by glucose from de novo synthesis of glycerolipids;Craven;Diabetes,1990

3. High glucose increases diacylglycerol mass and activates protein kinase C in mesangial cell cultures;Ayo;Am J Physiol,1991

4. Preferential elevation of protein kinase C isoform βII and diacylglycerol levels in the aorta and heart of diabetic rats: differential reversibility to glycemic control by islet cell transplantation;Inoguchi,1992

5. Insulin's effect on protein kinase C and diacylglycerol induced by diabetes and glucose in vascular tissues;Inoguchi;Am J Physiol,1994

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