Immunity and inflammation in diabetic kidney disease: translating mechanisms to biomarkers and treatment targets
Author:
Affiliation:
1. Division of Nephrology, University of Washington, Seattle, Washington;
2. Division of Nephrology, Kidney Research Institute, University of Washington, Seattle, Washington; and
3. Providence Health Care, Spokane, Washington
Abstract
Funder
HHS | NIH | National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
NHLBI
HHS | NIH | National Center for Advancing Translational Sciences (NCATS)
Patient-Centered Outcomes Research Institute (PCORI)
State of Washington
Publisher
American Physiological Society
Subject
Physiology
Link
https://www.physiology.org/doi/pdf/10.1152/ajprenal.00314.2016
Reference210 articles.
1. Nod-like Receptor Protein 3 (NLRP3) Inflammasome Activation and Podocyte Injury via Thioredoxin-Interacting Protein (TXNIP) during Hyperhomocysteinemia
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3. Functional diversity of helper T lymphocytes
4. Molecular basis for a link between complement and the vascular complications of diabetes
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