Interaction of Bordetella adenylate cyclase toxin with complement receptor 3 involves multivalent glycan binding-Reference-Cited by-同舟云学术

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Interaction of Bordetella adenylate cyclase toxin with complement receptor 3 involves multivalent glycan binding

Published:2014-12-29 Issue:3 Volume:589 Page:374-379
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Hasan Shakir,Osickova Adriana,Bumba Ladislav,Novák Petr,Sebo Peter,Osicka Radim

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/j.febslet.2014.12.023/fullpdf

Reference28 articles.

1. Pertussis toxin and extracytoplasmic adenylate cyclase as virulence factors of Bordetella pertussis;Weiss;J. Infect. Dis.,1984

2. Adenylate cyclase toxin is critical for colonization and pertussis toxin is critical for lethal infection by Bordetella pertussis in infant mice;Goodwin;Infect. Immun.,1990

3. The calmodulin-sensitive adenylate cyclase of Bordetella pertussis: cloning and expression in Escherichia coli;Glaser;Mol. Microbiol.,1988

4. The adenylate cyclase toxin of Bordetella pertussis binds to target cells via the alpha(M)beta(2) integrin (CD11b/CD18);Guermonprez;J. Exp. Med.,2001

5. Calmodulin activates prokaryotic adenylate cyclase;Wolff;Proc. Natl. Acad. Sci. USA,1980

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1. Membrane Interaction Characteristics of the RTX Toxins and the Cholesterol-Dependence of Their Cytolytic/Cytotoxic Activity;International Journal of Molecular Sciences;2024-03-08

2. A conserved tryptophan in the acylated segment of RTX toxins controls their β2 integrin–independent cell penetration;Journal of Biological Chemistry;2023-08

3. Interaction of RTX toxins with the host cell plasma membrane;Biological Chemistry;2023-03-14

4. Structural basis for non-canonical integrin engagement by Bordetella adenylate cyclase toxin;Cell Reports;2022-08

5. Pertussis toxin suppresses dendritic cell-mediated delivery of B. pertussis into lung-draining lymph nodes;PLOS Pathogens;2022-06-06

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