Protein kinase Cθ gene expression is oppositely regulated by GCN5 and EBF1 in immature B cells-Reference-Cited by-同舟云学术

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Protein kinase Cθ gene expression is oppositely regulated by GCN5 and EBF1 in immature B cells

Published:2014-03-19 Issue:9 Volume:588 Page:1739-1742
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:

Author:

Kikuchi Hidehiko,Nakayama Masami,Kuribayashi Futoshi,Imajoh-Ohmi Shinobu,Nishitoh Hideki,Takami Yasunari,Nakayama Tatsuo

Funder

Scientific Research from the Ministry of Education, Culture, Sports, Science and Technology of Japan

Joint Research Project of the Institute of Medical Science

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1016/j.febslet.2014.03.025/fullpdf

Reference30 articles.

1. Protein kinase C and lipid signaling for sustained cellular responses;Nishizuka;FASEB J.,1995

2. Protein kinase C isotypes and their specific functions: prologue;Ohno;J. Biochem.,2002

3. Protein kinase C (PKC) isozyme-specific substrates and their design;Kang;Biotechnol. Adv.,2012

4. High resolution immunogold analysis reveals distinct subcellular compartmentation of protein kinase Cγ and δ in rat Purkinje cells;Cardell;Neuroscience,1998

5. A Ca2+-independent protein kinase C, nPKCη: its structure, distribution and possible function;Hashimono;Tohoku J. Exp. Med.,1992

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1. GCN5-deficiency remarkably enhances the sensitivity of B cells in response to 4-nitroquinoline 1-oxide;Fundamental Toxicological Sciences;2016

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