Indirubin Derivatives Modulate TGFβ/BMP Signaling at Different Levels and Trigger Ubiquitin-Mediated Depletion of Nonactivated R-Smads-Reference-Cited by-同舟云学术

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Indirubin Derivatives Modulate TGFβ/BMP Signaling at Different Levels and Trigger Ubiquitin-Mediated Depletion of Nonactivated R-Smads

Published:2012-11 Issue:11 Volume:19 Page:1423-1436
ISSN:1074-5521
Container-title:Chemistry & Biology
language:en
Short-container-title:Chemistry & Biology

Author:

Cheng Xinlai,Alborzinia Hamed,Merz Karl-Heinz,Steinbeisser Herbert,Mrowka Ralf,Scholl Catharina,Kitanovic Igor,Eisenbrand Gerhard,Wölfl Stefan

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Drug Discovery,Pharmacology,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Reference53 articles.

1. Connective-tissue growth factor (CTGF) modulates cell signalling by BMP and TGF-β;Abreu;Nat. Cell Biol.,2002

2. Nuclear CDKs drive Smad transcriptional activation and turnover in BMP and TGF-β pathways;Alarcón;Cell,2009

3. The protein Id: a negative regulator of helix-loop-helix DNA binding proteins;Benezra;Cell,1990

4. The homeobox gene Siamois is a target of the Wnt dorsalisation pathway and triggers organiser activity in the absence of mesoderm;Carnac;Development,1996

5. Synthesis and cytotoxicity of novel indirubin-5-carboxamides;Cheng;Bioorg. Med. Chem.,2010

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