Mouse models of Parkinson's disease associated with mitochondrial dysfunction-Reference-Cited by-同舟云学术

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Mouse models of Parkinson's disease associated with mitochondrial dysfunction

Published:2013-07 Issue: Volume:55 Page:87-94
ISSN:1044-7431
Container-title:Molecular and Cellular Neuroscience
language:en
Short-container-title:Molecular and Cellular Neuroscience

Author:

Pickrell Alicia M.,Pinto Milena,Moraes Carlos T.

Funder

National Institutes of Health

Muscular Dystrophy Association

5T32NS007492

5T32NS007459

American Heart Association

Lois Pope LIFE Fellowship

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience,Molecular Biology

Reference108 articles.

1. Mice lacking alpha-synuclein display functional deficits in the nigrostriatal dopamine system;Abeliovich;Neuron,2000

2. Increased mitochondrial calcium sensitivity and abnormal expression of innate immunity genes precede dopaminergic defects in Pink1-deficient mice;Akundi;PLoS One,2011

3. DJ-1 protects the nigrostriatal axis from the neurotoxin MPTP by modulation of the AKT pathway;Aleyasin;Proc. Natl. Acad. Sci. U. S. A.,2010

4. Engrailed protects mouse midbrain dopaminergic neurons against mitochondrial complex I insults;Alvarez-Fischer;Nat. Neurosci.,2011

5. Sequence and organization of the human mitochondrial genome;Anderson;Nature,1981

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1. Genetic lesions of the noradrenergic system trigger induction of oxidative stress and inflammation in the ventral midbrain;Neurochemistry International;2022-05

2. Parkinsons Disease-Like Neuropathology and Phenotype Following Induction of Oxidative Stress and Inflammation in the Brain;Journal of Biomedical Research & Environmental Sciences;2022-01

3. Comprehensive Perspectives on Experimental Models for Parkinson’s Disease;Aging and disease;2021

4. Mitochondrial Dysfunction and PINK1Gene Mutation: In Parkinson‘sDisease (PD);INTERNATIONAL JOURNAL OF HUMAN GENETICS;2020-07-05

5. Increased plasma phosphatidylcholine/lysophosphatidylcholine ratios in patients with Parkinson's disease;Rapid Communications in Mass Spectrometry;2020-01-15

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