Wild type and P301L mutant Tau promote neuro-inflammation and α-Synuclein accumulation in lentiviral gene delivery models-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

Wild type and P301L mutant Tau promote neuro-inflammation and α-Synuclein accumulation in lentiviral gene delivery models

Published:2012-01 Issue:1 Volume:49 Page:44-53
ISSN:1044-7431
Container-title:Molecular and Cellular Neuroscience
language:en
Short-container-title:Molecular and Cellular Neuroscience

Author:

Khandelwal Preeti J.,Dumanis Sonya B.,Herman Alexander M.,Rebeck G. William,Moussa Charbel E.-H.

Publisher

Elsevier BV

Subject

Cell Biology,Cellular and Molecular Neuroscience,Molecular Biology

Reference51 articles.

1. Association of an extended haplotype in the tau gene with progressive supranuclear palsy;Baker;Hum. Mol. Genet.,1999

2. Accumulation of abnormally phosphorylated tau precedes the formation of neurofibrillary tangles in Alzheimer's disease;Bancher;Brain Res.,1989

3. Vulnerability of cortical neurons to Alzheimer's and Parkinson's diseases;Braak;J. Alzheimers Dis.,2006

4. Tau protein isoforms, phosphorylation and role in neurodegenerative disorders;Buee;Brain Res. Brain Res. Rev.,2000

5. Parkin promotes intracellular Abeta1-42 clearance;Burns;Hum. Mol. Genet.,2009

Cited by 49 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Multivalency drives interactions of alpha-synuclein fibrils with tau;PLOS ONE;2024-09-10

2. Alzheimer’s disease and neuroinflammation: will new drugs in clinical trials pave the way to a multi-target therapy?;Frontiers in Pharmacology;2023-06-02

3. Erratum to “wild type and P301L mutant Tau promote neuro-inflammation and α-Synuclein accumulation in lentiviral gene delivery models” [Mol. Cell. Neurosci. 49 (1) (2012), 44–53];Molecular and Cellular Neuroscience;2023-06

4. Inhibition of PLK2 activity affects APP and tau pathology and improves synaptic content in a sex-dependent manner in a 3xTg mouse model of Alzheimer's disease;Neurobiology of Disease;2022-10

5. Arc Regulates Transcription of Genes for Plasticity, Excitability and Alzheimer’s Disease;Biomedicines;2022-08-11

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3