The effects of type 1 IGF receptor inhibition in a mouse model of diabetic kidney disease
Author:
Publisher
Elsevier BV
Subject
Endocrinology,Endocrinology, Diabetes and Metabolism
Reference34 articles.
1. Novel roles of the IGF–IGFBP axis in etiopathophysiology of diabetic nephropathy;Vasylyeva;Diabetes Res. Clin. Pract.,2007
2. Growth hormone, the insulin-like growth factor system, and the kidney;Feld;Endocr. Rev.,1996
3. A novel somatostatin analogue prevents early renal complications in the nonobese diabetic mouse;Landau;Kidney Int.,2001
4. Contribution of growth hormone and IGF-I to early diabetic nephropathy in type 1 diabetes;Cummings;Diabetes,1998
5. Increased renal Akt/mTOR and MAPK signaling in type I diabetes in the absence of IGF type 1 receptor activation;Landau;Endocrine,2009
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Human umbilical cord mesenchymal stem cells attenuate diabetic nephropathy through the IGF1R-CHK2-p53 signalling axis in male rats with type 2 diabetes mellitus;Journal of Zhejiang University-SCIENCE B;2024-07
2. IGF-1/IGF-1R blockade ameliorates diabetic kidney disease through normalizing Snail1 expression in a mouse model;American Journal of Physiology-Endocrinology and Metabolism;2019-10-01
3. IgA Enhances IGF-1 Mitogenic Activity Via Receptor Modulation in Glomerular Mesangial Cells: Implications for IgA-Induced Nephropathy;Kidney and Blood Pressure Research;2017
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