Pancreatic antigenic complex p64 69: involvement in regulation of insulin secretion and relation to glutamic acid decarboxylase
Author:
Publisher
Elsevier BV
Subject
Immunology,Immunology and Allergy
Reference30 articles.
1. Type I diabetes mellitus: A predictable autoimmune disease with interindividual variation in the rate of β cell destruction
2. Perspectives on the molecular biology and immunology of the pancreatic β cells;Baekkeskov,1989
3. Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase
4. Pancreatic Islet Cells in Tissue Culture: Function and Immunoreactivity with Serum Autoantibodies from Diabetes Type-1 Patients and Monoclonal Antibodies ICA-1
Cited by 5 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Response of glutamic acid decarboxylase to glucose but not arginine in islets;Life Sciences;1995-04
2. A convulsant, 3-mercaptopropionic acid, decreases the level of GABA and GAD in rat pancreatic islets and brain;Experientia;1995-03
3. The Effect of Cytokines on Expression of Glutamic Acid Decarboxylase-65 in Cultured Islets;Autoimmunity;1995-01
4. Functional State of the β Cell Affects Expression of Both Forms of Glutamic Acid Decarboxylase;Pancreas;1994-09
5. Recombinant glutamic acid decarboxylase (representing the single isoform expressed in human islets) detects IDDM-associated 64,000-M(r) autoantibodies;Diabetes;1992-10-01
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