Synthetic Lethality of Combined Bcl-2 Inhibition and p53 Activation in AML: Mechanisms and Superior Antileukemic Efficacy-Reference-Cited by-同舟云学术

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Synthetic Lethality of Combined Bcl-2 Inhibition and p53 Activation in AML: Mechanisms and Superior Antileukemic Efficacy

Published:2017-12 Issue:6 Volume:32 Page:748-760.e6
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Pan Rongqing,Ruvolo Vivian,Mu Hong,Leverson Joel D.,Nichols Gwen,Reed John C.,Konopleva Marina,Andreeff Michael

Funder

NIH

MD Anderson's Cancer Center

Paul and Mary Haas Chair

UT Health Science Center

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference38 articles.

1. Small molecule Mcl-1 inhibitors for the treatment of cancer;Belmar;Pharmacol. Ther.,2015

2. The cBio cancer genomics portal: an open platform for exploring multidimensional cancer genomics data;Cerami;Cancer Discov.,2012

3. Negative feedback and adaptive resistance to the targeted therapy of cancer;Chandarlapaty;Cancer Discov.,2012

4. p53 levels, functional domains, and DNA damage determine the extent of the apoptotic response of tumor cells;Chen;Genes Dev.,1996

5. Detection of protein ubiquitination;Choo;J. Vis. Exp.,2009

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