Histone H3.3 K27M Accelerates Spontaneous Brainstem Glioma and Drives Restricted Changes in Bivalent Gene Expression-Reference-Cited by-同舟云学术

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Histone H3.3 K27M Accelerates Spontaneous Brainstem Glioma and Drives Restricted Changes in Bivalent Gene Expression

Published:2019-01 Issue:1 Volume:35 Page:140-155.e7
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Larson Jon D.,Kasper Lawryn H.,Paugh Barbara S.,Jin Hongjian,Wu Gang,Kwon Chang-Hyuk,Fan Yiping,Shaw Timothy I.,Silveira André B.,Qu Chunxu,Xu Raymond,Zhu Xiaoyan,Zhang Junyuan,Russell Helen R.,Peters Jennifer L.,Finkelstein David,Xu Beisi,Lin Tong,Tinkle Christopher L.,Patay Zoltan,Onar-Thomas Arzu,Pounds Stanley B.,McKinnon Peter J.,Ellison David W.,Zhang Jinghui,Baker Suzanne J.

Funder

NIH

Swim Across America

NCI

Musicians Against Childhood Cancer

ALSAC

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference58 articles.

1. HTSeq–a Python framework to work with high-throughput sequencing data;Anders;Bioinformatics,2015

2. Lowered H3K27me3 and DNA hypomethylation define poorly prognostic pediatric posterior fossa ependymomas;Bayliss;Sci. Transl Med.,2016

3. Reduced H3K27me3 and DNA hypomethylation are major drivers of gene expression in K27M mutant pediatric high-grade gliomas;Bender;Cancer Cell,2013

4. Genomic analysis of diffuse intrinsic pontine gliomas identifies three molecular subgroups and recurrent activating ACVR1 mutations;Buczkowicz;Nat. Genet.,2014

5. The histone H3.3K27M mutation in pediatric glioma reprograms H3K27 methylation and gene expression;Chan;Genes Dev.,2013

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1. Histone H3 mutations and their impact on genome stability maintenance;Biochemical Society Transactions;2024-09-09

2. A pontine-specific axonal niche supports de novo gliomagenesis;2024-09-07

3. H3K27M diffuse midline glioma is homologous recombination defective and sensitized to radiotherapy and NK cell-mediated antitumor immunity by PARP inhibition;2024-08-27

4. Brain Stem Glioma Recurrence: Exploring the Therapeutic Frontiers;Journal of Personalized Medicine;2024-08-25

5. Dominant Malignant Clones Leverage Lineage Restricted Epigenomic Programs to Drive Ependymoma Development;2024-08-15

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