Late effect of developmental exposure to glycidol on hippocampal neurogenesis in mice: Loss of parvalbumin-expressing interneurons-Reference-Cited by-同舟云学术

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Late effect of developmental exposure to glycidol on hippocampal neurogenesis in mice: Loss of parvalbumin-expressing interneurons

Published:2017-09 Issue:7 Volume:69 Page:517-526
ISSN:0940-2993
Container-title:Experimental and Toxicologic Pathology
language:en
Short-container-title:Experimental and Toxicologic Pathology

Author:

Kawashima Masashi,Watanabe Yousuke,Nakajima Kota,Murayama Hirotada,Nagahara Rei,Jin Meilan,Yoshida Toshinori,Shibutani Makoto

Funder

Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Cell Biology,Toxicology,General Medicine,Pathology and Forensic Medicine

Reference41 articles.

1. Methacarn as a whole brain fixative for gene and protein expression analyses of specific brain regions in rats;Akane;J. Toxicol. Sci.,2013

2. Glycidol induces axonopathy by adult-stage exposure and aberration of hippocampal neurogenesis affecting late-stage differentiation by developmental exposure in rats;Akane;Toxicol. Sci.,2013

3. Glycidol induces axonopathy and aberrations of hippocampal neurogenesis affecting late-stage differentiation by exposure to rats in a framework of 28-day toxicity study;Akane;Toxcol. Lett.,2014

4. Regulation of adult neurogenesis by excitatory input and NMDA receptor activation in the dentate gyrus;Cameron;J. Neurosci.,1995

5. The impact of NMDA receptor hypofunction on GABAergic neurons in the pathophysiology of schizophrenia;Cohen;Schizophr. Res.,2015

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