ES cells do not activate p53-dependent stress responses and undergo p53-independent apoptosis in response to DNA damage-Reference-Cited by-同舟云学术

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ES cells do not activate p53-dependent stress responses and undergo p53-independent apoptosis in response to DNA damage

Published:1998-01 Issue:3 Volume:8 Page:145-155
ISSN:0960-9822
Container-title:Current Biology
language:en
Short-container-title:Current Biology

Author:

Aladjem Mirit I.,Spike Benjamin T.,Rodewald Luo Wei,Hope Thomas J.,Klemm Martina,Jaenisch Rudolf,Wahl Geoffrey M.

Publisher

Elsevier BV

Subject

General Agricultural and Biological Sciences,General Biochemistry, Genetics and Molecular Biology

Reference79 articles.

1. Transient hypoxia enhances the frequency of dihydrofolate reductase gene amplification in Chinese hamster ovary cells;Rice;Proc Natl Acad Sci USA,1986

2. Altered cell cycle arrest and gene amplification potential accompany loss of wild-type p53;Livingstone;Cell,1992

3. Wild-type p53 restores cell cycle control and inhibits gene amplification in cells with mutant p53 alleles;Yin;Cell,1992

4. Enhancement of methotrexate resistance and dihydrofolate reductase gene amplification by treatment of mouse 3T6 cells with hydroxyurea;Brown;Mol Cell Biol,1983

5. Checkpoints: controls that ensure the order of cell cycle events;Hartwell;Science,1989

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1. DNA damage induces p53-independent apoptosis through ribosome stalling;Science;2024-05-17

2. A dynamic role for transcription factors in restoring transcription through mitosis;Biochemical Society Transactions;2024-03-25

3. Transcriptome Analysis by RNA Sequencing of Mouse Embryonic Stem Cells Stocked on International Space Station for 1584 Days in Frozen State after Culture on the Ground;International Journal of Molecular Sciences;2024-03-14

4. Aberrant ATM signaling and homology-directed DNA repair as a vulnerability of p53-mutant GBM to AZD1390-mediated radiosensitization;Science Translational Medicine;2024-02-14

5. A common cellular response to broad splicing perturbations is characterized by metabolic transcript downregulation driven by the Mdm2–p53 axis;Disease Models & Mechanisms;2024-02-01

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