NLRC5 negatively regulates inflammatory responses in LPS-induced acute lung injury through NF-κB and p38 MAPK signal pathways-Reference-Cited by-同舟云学术

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NLRC5 negatively regulates inflammatory responses in LPS-induced acute lung injury through NF-κB and p38 MAPK signal pathways

Published:2020-09 Issue: Volume:403 Page:115150
ISSN:0041-008X
Container-title:Toxicology and Applied Pharmacology
language:en
Short-container-title:Toxicology and Applied Pharmacology

Author:

Wang Yuanyuan,Huang Cheng,Bian Erbao,Lei Ting,Lv Xiongwen,Li Jun

Funder

National Natural Science Foundation of China

University Synergy Innovation Program of Anhui Province

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference35 articles.

1. Diverse macrophage populations mediate acute lung inflammation and resolution;Aggarwal;Am. J. Physiol.,2014

2. Angiotensin-converting enzyme 2 inhibits apoptosis of pulmonary endothelial cells during acute lung injury through suppressing MiR-4262;Bao;Cell. Physiol. Biochem.,2015

3. Repeated Domperidone treatment modulates pulmonary cytokines in LPS-induced acute lung injury in mice;Barreto;Int. Immunopharmacol.,2018

4. Incidence of acute respiratory distress syndrome—reply;Bellani;JAMA,2016

5. Noninvasive ventilation of patients with acute respiratory distress syndrome. Insights from the LUNG SAFE study;Bellani;Am. J. Respir. Crit. Care Med.,2017

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4. PROTECTIVE MECHANISM OF ALLEVIATING LUNG INJURY BY INHIBITING NF-κB PATHWAY;Journal of Mechanics in Medicine and Biology;2023-10-11

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