The critical role for TAK1 in trichloroethylene-induced contact hypersensitivity in vivo and in CD4+ T cell function alteration by trichloroethylene and its metabolites in vitro
Author:
Funder
National Natural Science Foundation of China
Publisher
Elsevier BV
Subject
Pharmacology,Toxicology
Reference52 articles.
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2. Cell type-specific function of tak1 in innate immune signaling;Ajibade;Trends Immunol.,2013
3. Contact hypersensitivity models in mice;Allen;Methods Mol. Biol.,2013
4. The llna: a brief review of recent advances and limitations;Anderson;J. Allergy,2011
5. Activation and attenuation of apoptosis of cd4+ t cells following in vivo exposure to two common environmental toxicants, trichloroacetaldehyde hydrate and trichloroacetic acid;Blossom;J. Autoimmun.,2004
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. ROS-mediated inflammatory response in liver damage via regulating the Nrf2/HO-1/NLRP3 pathway in mice with trichloroethylene hypersensitivity syndrome;Journal of Immunotoxicology;2022-09-07
2. Glomerular Damage in Trichloroethylene-Sensitized Mice: Targeting Cathepsin L-Induced Hyperactive mTOR Signaling;Frontiers in Pharmacology;2021-07-29
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