ROS-mediated inflammatory response in liver damage via regulating the Nrf2/HO-1/NLRP3 pathway in mice with trichloroethylene hypersensitivity syndrome-Reference-Cited by-同舟云学术

ROS-mediated inflammatory response in liver damage via regulating the Nrf2/HO-1/NLRP3 pathway in mice with trichloroethylene hypersensitivity syndrome

Published:2022-09-07 Issue:1 Volume:19 Page:100-108
ISSN:1547-691X
Container-title:Journal of Immunotoxicology
language:en
Short-container-title:Journal of Immunotoxicology

Author:

Wang Feng¹²^ORCID,Hong Yiting³,Jiang Wei³,Wang Yican³,Chen Muyue³,Zang Dandan⁴,Zhu Qixing²⁵

Affiliation:

1. Department of Dermatology, Second Hospital of Anhui Medical University, Hefei, China

2. Key Laboratory of Dermatology, Ministry of Education, First Affiliated Hospital of Anhui Medical University, Hefei, China

3. Department of Occupational Health and Environmental Health, School of Public Health, Anhui Medical University, Hefei, China

4. Center for Scientific Research and Experiment, Anhui Medical University, Hefei, China

5. Department of Dermatology, First Affiliated Hospital of Anhui Medical University, Hefei, China

Funder

National Natural Science Foundation of China

Scientific Research Foundation of Anhui Medical University

Publisher

Informa UK Limited

Subject

Toxicology,Immunology

Link

https://www.tandfonline.com/doi/pdf/10.1080/1547691X.2022.2111003

Reference25 articles.

1. Environmentally toxicant exposures induced intragenerational transmission of liver abnormalities in mice

2. Enhancing the Nrf2 Antioxidant Signaling Provides Protection Against Trichloroethene-mediated Inflammation and Autoimmune Response

3. Reactive Oxygen Stimulation of Interleukin-6 Release in the Human Trophoblast Cell Line HTR-8/SVneo by the Trichlorethylene Metabolite S-(1,2-Dichloro)-L-Cysteine

4. Tempol treatment shows phenotype improvement in mdx mice

5. Impairment of type H vessels by NOX2-mediated endothelial oxidative stress: critical mechanisms and therapeutic targets for bone fragility in streptozotocin-induced type 1 diabetic mice

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