The role of the hemostatic system in murine liver injury induced by coexposure to lipopolysaccharide and trovafloxacin, a drug with idiosyncratic liability
Author:
Publisher
Elsevier BV
Subject
Pharmacology,Toxicology
Cited by 25 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. What have we learned from animal models of idiosyncratic, drug-induced liver injury?;Expert Opinion on Drug Metabolism & Toxicology;2020-05-04
2. Dibromoacetic Acid Induced Hepatotoxicity in Mice through Oxidative Stress and Toll-Like Receptor 4 Signaling Pathway Activation;Oxidative Medicine and Cellular Longevity;2019-11-20
3. Functional modulation of liver mitochondria in lipopolysaccharide/drug co-treated rat liver injury model;The Journal of Toxicological Sciences;2019
4. Trovafloxacin-Induced Liver Injury: Lack in Regulation of Inflammation by Inhibition of Nucleotide Release and Neutrophil Movement;Toxicological Sciences;2018-09-22
5. Tissue influx of neutrophils and monocytes is delayed during development of trovafloxacin-induced tumor necrosis factor-dependent liver injury in mice;Journal of Applied Toxicology;2018-01-26
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