Nitric oxide production by mouse renal tubules can be increased by a sodium-dependent mechanism

Author:

Kempson Stephen,Thompson Nathan,Pezzuto Laura,Glenn Bohlen H.

Publisher

Elsevier BV

Subject

Cancer Research,Clinical Biochemistry,Physiology,Biochemistry

Reference45 articles.

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2. Mechanism of increased vessel wall nitric oxide concentrations during intestinal absorption;Bohlen;Am. J. Physiol.,1998

3. Sodium channels are required during in vivo sodium chloride hyperosmolarity to stimulate an increase in intestinal endothelial nitric oxide production;Zani;Am. J. Physiol. Heart Circ. Physiol.,2005

4. Sodium hyperosmolarity of intestinal lymph causes arteriolar vasodilation in part mediated by EDRF;Steenbergen;Am. J. Physiol.,1993

5. Role of iNOS and eNOS in modulating proximal tubule transport and acid-base balance;Wang;Am. J. Physiol. Renal Physiol.,2002

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