Absence of SOD1 leads to oxidative stress in peripheral nerve and causes a progressive distal motor axonopathy-Reference-Cited by-同舟云学术

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Absence of SOD1 leads to oxidative stress in peripheral nerve and causes a progressive distal motor axonopathy

Published:2012-01 Issue:1 Volume:233 Page:163-171
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Fischer Lindsey R.,Li Yingjie,Asress Seneshaw A.,Jones Dean P.,Glass Jonathan D.

Funder

Robert Packard Center for ALS Research

NIH

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference47 articles.

1. Overexpression of the neural growth-associated protein GAP-43 induces nerve sprouting in the adult nervous system of transgenic mice;Aigner;Cell,1995

2. Oxidative stress in ALS: a mechanism of neurodegeneration and a therapeutic target;Barber;Biochim. Biophys. Acta (BBA) Mol. Basis Dis.,2006

3. Botulinum neurotoxin A selectively cleaves the synaptic protein SNAP-25;Blasi;Nature,1993

4. ALS: a disease of motor neurons and their nonneuronal neighbors;Boillée;Neuron,2006

5. Superoxide dismutase 1 with mutations linked to familial amyotrophic lateral sclerosis possesses significant activity;Borchelt;Proc. Natl. Acad. Sci. U. S. A.,1994

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