Mitochondria-associated microRNAs in rat hippocampus following traumatic brain injury-Reference-Cited by-同舟云学术

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Mitochondria-associated microRNAs in rat hippocampus following traumatic brain injury

Published:2015-03 Issue: Volume:265 Page:84-93
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Wang Wang-Xia,Visavadiya Nishant P.,Pandya Jignesh D.,Nelson Peter T.,Sullivan Patrick G.,Springer Joe E.

Funder

Morton Cure Paralysis Fund

Cardinal Hill Rehabilitation Hospital

National Center for Advancing Translational Sciences

National Institutes of Health

PHS

KSCHIRT

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference84 articles.

1. Oxidative stress and modification of synaptic proteins in hippocampus after traumatic brain injury;Ansari;Free Radic. Biol. Med.,2008

2. Loss of m-AAA protease in mitochondria causes complex I deficiency and increased sensitivity to oxidative stress in hereditary spastic paraplegia;Atorino;J. Cell Biol.,2003

3. Neuronal cell loss in the CA3 subfield of the hippocampus following cortical contusion utilizing the optical disector method for cell counting;Baldwin;J. Neurotrauma,1997

4. Nuclear outsourcing of RNA interference components to human mitochondria;Bandiera;PLoS One,2011

5. Pre-microRNA and mature microRNA in human mitochondria;Barrey;PLoS One,2011

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