A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons-Reference-Cited by-同舟云学术

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A sodium channel mutation linked to epilepsy increases ramp and persistent current of Nav1.3 and induces hyperexcitability in hippocampal neurons

Published:2010-08 Issue:2 Volume:224 Page:362-368
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Estacion Mark,Gasser Andreas,Dib-Hajj Sulayman D.,Waxman Stephen G.

Funder

Medical Research Service and Rehabilitation Research Service, Dept. of Veterans Affairs

National Multiple Sclerosis Society

Erythromelalgia Association

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference41 articles.

1. Postnatal development of a persistent Na+ current in pyramidal neurons from rat sensorimotor cortex;Alzheimer;J. Neurophysiol.,1993

2. Ionic basis of spike after-depolarization and burst generation in adult rat hippocampal CA1 pyramidal cells;Azouz;J. Physiol.,1996

3. GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones;Baker;J. Physiol. (Lond.),2003

4. Differential regulation of three sodium channel messenger RNAs in the rat central nervous system during development;Beckh;EMBO J.,1989

5. Spinal sensory neurons express multiple sodium channel alpha-subunit mRNAs;Black;Mol. Brain Res.,1996

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