Sustained levels of antibodies against Aβ in amyloid-rich regions of the CNS following intravenous dosing in human APP transgenic mice-Reference-Cited by-同舟云学术

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Sustained levels of antibodies against Aβ in amyloid-rich regions of the CNS following intravenous dosing in human APP transgenic mice

Published:2012-11 Issue:1 Volume:238 Page:38-43
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Bard Frédérique,Fox Michael,Friedrich Stuart,Seubert Peter,Schenk Dale,Kinney Gene G.,Yednock Ted

Funder

Elan Pharmaceuticals, Inc.

Wyeth Research

Pfizer Inc.

Janssen Alzheimer Immunotherapy Research & Development, LLC

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference16 articles.

1. Passage of amyloid b protein antibody across the blood–brain barrier in a mouse model of Alzheimer's disease;Banks;Peptides,2002

2. Peripherally administered antibodies against amyloid β-peptide enter the central nervous system and reduce pathology in a mouse model of Alzheimer disease;Bard;Nat. Med.,2000

3. Gantenerumab: a novel human anti-Aβ antibody demonstrates sustained cerebral amyloid-β binding and elicits cell-mediated removal of human amyloid-β;Bohrman;J. Alzheimers Dis.,2012

4. Serum proteins bypass the blood–brain fluid barriers for extracellular entry to the central nervous system;Broadwell;Exp. Neurol.,1993

5. Amyloid precursor protein processing and Aβ42 deposition in a transgenic mouse model of Alzheimer disease;Johnson-Wood;Proc. Natl. Acad. Sci. U. S. A.,1997

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