Improvement of the skeletal and dental hypophosphatasia phenotype in Alpl−/− mice by administration of soluble (non-targeted) chimeric alkaline phosphatase-Reference-Cited by-同舟云学术

Improvement of the skeletal and dental hypophosphatasia phenotype in Alpl−/− mice by administration of soluble (non-targeted) chimeric alkaline phosphatase

Published:2015-03 Issue: Volume:72 Page:137-147
ISSN:8756-3282
Container-title:Bone
language:en
Short-container-title:Bone

Author:

Gasque Kellen C.S.,Foster Brian L.,Kuss Pia,Yadav Manisha C.,Liu Jin,Kiffer-Moreira Tina,van Elsas Andrea,Hatch Nan,Somerman Martha J.,Millán José Luis

Funder

National Institutes of Health

AM-Pharma

National Institute of Arthritis and Musculoskeletal and Skin Diseases

Publisher

Elsevier BV

Subject

Histology,Physiology,Endocrinology, Diabetes and Metabolism

Reference59 articles.

1. Impaired calcification around matrix vesicles of growth plate and bone in alkaline phosphatase-deficient mice;Anderson;Am J Pathol,2004

2. Sustained osteomalacia of long bones despite major improvement in other hypophosphatasia-related mineral deficits in tissue nonspecific alkaline phosphatase/nucleotide pyrophosphatase phosphodiesterase 1 double-deficient mice;Anderson;Am J Pathol,2005

3. Pyridoxine-responsive seizures as the first symptom of infantile hypophosphatasia caused by two novel missense mutations (c.677T>C, p.M226T; c.1112C>T, p.T371I) of the tissue-nonspecific alkaline phosphatase gene;Baumgartner-Sigl;Bone,2007