FGF23 is processed by proprotein convertases but not by PHEX-Reference-Cited by-同舟云学术

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FGF23 is processed by proprotein convertases but not by PHEX

Published:2004-08 Issue:2 Volume:35 Page:455-462
ISSN:8756-3282
Container-title:Bone
language:en
Short-container-title:Bone

Author:

Benet-Pagès Anna,Lorenz-Depiereux Bettina,Zischka Hans,White Kenneth E,Econs Michael J,Strom Tim M

Publisher

Elsevier BV

Subject

Histology,Physiology,Endocrinology, Diabetes and Metabolism

Reference42 articles.

1. Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23;White;Kidney Int.,2001

2. Subtilase-like pro-protein convertases: from molecular specificity to therapeutic applications;Bergeron;J. Mol. Endocrinol.,2000

3. Proprotein and prohormone convertases: a family of subtilases generating diverse bioactive polypeptides;Seidah;Brain Res.,1999

4. Furin at the cutting edge: from protein traffic to embryogenesis and disease;Thomas;Nat. Rev., Mol. Cell Biol.,2002

5. Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23. The ADHR Consortium;The ADHR Consortium;Nat. Genet.,2000

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4. Complex intrinsic abnormalities in osteoblast lineage cells of X-linked hypophosphatemia: Analysis of human iPS cell models generated by CRISPR/Cas9-mediated gene ablation;Bone;2024-04

5. A Novel Pathogenic Variant in <i>Fibroblast Growth Factor 23</i> outside the Furin-Recognizing RXXR Motif in an Autosomal Dominant Hypophosphatemic Rickets Patient;Hormone Research in Paediatrics;2024-03-15

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