Association between Oxidative Stress, Genetic Factors, and Clinical Severity in Children with Sickle Cell Anemia

Author:

Renoux Céline,Joly Philippe,Faes Camille,Mury Pauline,Eglenen Buse,Turkay Mine,Yavas Gokce,Yalcin Ozlem,Bertrand Yves,Garnier Nathalie,Cuzzubbo Daniela,Gauthier Alexandra,Romana Marc,Möckesch Berenike,Cannas Giovanna,Antoine-Jonville Sophie,Pialoux Vincent,Connes PhilippeORCID

Publisher

Elsevier BV

Subject

Pediatrics, Perinatology and Child Health

Reference66 articles.

1. Sickle-cell disease;Stuart;Lancet,2004

2. Effect of alpha-globin genotype on the pathophysiology of sickle cell disease;Ballas;Pediatr Pathol Mol Med,2001

3. G6PD deficiency, absence of α-thalassemia, and hemolytic rate at baseline are significant independent risk factors for abnormally high cerebral velocities in patients with sickle cell anemia;Bernaudin;Blood,2008

4. Evidence that microdeletions in the α globin gene protect against the development of sickle cell glomerulopathy in humans;Guasch;J Am Soc Nephrol,1999

5. The interaction of alpha-thalassemia and homozygous sickle-cell disease;Higgs;N Engl J Med,1982

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