Mitochondrial diseases: Yeast as a model for the study of suppressors
Author:
Funder
Pasteur Institute Italy - Cenci Bolognetti Foundation
Sapienza University of Rome
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference35 articles.
1. The yeast counterparts of human ‘MELAS’ mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF-Tu;Feuermann;EMBO Rep.,2003
2. Mutations in yeast mt tRNAs: specific and general suppression by nuclear encoded tRNA interactors;De Luca;Gene,2006
3. Yeast as a model of human mitochondrial tRNA base substitutions: investigation of the molecular basis of respiratory defects;Montanari;RNA,2008
4. Can yeast be used to study mitochondrial diseases? Biolistic tRNA mutants for the analysis of mechanisms and suppressors;De Luca;Mitochondrion,2009
5. Aminoacyl-tRNA synthetases are multivalent suppressors of defects due to human equivalent mutations in yeast mt tRNA genes;Montanari;Biochim. Biophys. Acta,2010
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2. The Power of Yeast in Modelling Human Nuclear Mutations Associated with Mitochondrial Diseases;Genes;2021-02-20
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