The yeast counterparts of human ‘MELAS’ mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF‐Tu-Reference-Cited by-同舟云学术

The yeast counterparts of human ‘MELAS’ mutations cause mitochondrial dysfunction that can be rescued by overexpression of the mitochondrial translation factor EF‐Tu

Published:2003-01 Issue:1 Volume:4 Page:53-58
ISSN:1469-221X
Container-title:EMBO reports
language:en
Short-container-title:EMBO Rep

Author:

Feuermann M¹,Francisci S²,Rinaldi T²,De Luca C²,Rohou H¹,Frontali L²,Bolotin‐Fukuhara M¹

Affiliation:

1. Laboratoire de Génétique Moléculaire, Institut de Génétique et Microbiologie, Bâtiment 400, Université Paris Sud 91405 Orsay Cedex France

2. Pasteur Institute—Cenci Bolognetti Foundation, Department of Cell and Developmental Biology, University of Rome I ‘La Sapienza’ Piazzale Aldo Moro 5 I‐00185 Rome Italy

Publisher

EMBO

Subject

Genetics,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1038/sj.embor.embor713

Reference20 articles.

1. Genetic transformation of Saccharomyces cerevisiae mitochondria

2. CONTROL OF MITOCHONDRIAL GENE EXPRESSION IN SACCHAROMYCES CEREVISIAE

3. [10] Analysis and manipulation of yeast mitochondrial genes

4. Ts mutations in mitochondrial tRNA genes: characterization and effects of two point mutations in the mitochondrial gene for tRNAphe in Saccharomyces cerevisiae

5. A Set of Vectors with a Tetracycline-Regulatable Promoter System for Modulated Gene Expression inSaccharomyces cerevisiae

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