Severe protein aggregate myopathy in a knockout mouse model points to an essential role of cofilin2 in sarcomeric actin exchange and muscle maintenance-Reference-Cited by-同舟云学术

Severe protein aggregate myopathy in a knockout mouse model points to an essential role of cofilin2 in sarcomeric actin exchange and muscle maintenance

Published:2014-05 Issue:5-6 Volume:93 Page:252-266
ISSN:0171-9335
Container-title:European Journal of Cell Biology
language:en
Short-container-title:European Journal of Cell Biology

Author:

Gurniak Christine B.,Chevessier Frédéric,Jokwitz Melanie,Jönsson Friederike,Perlas Emerald,Richter Hendrik,Matern Gabi,Boyl Pietro Pilo,Chaponnier Christine,Fürst Dieter,Schröder Rolf,Witke Walter

Funder

EMBL

DFG

Publisher

Elsevier BV

Subject

Cell Biology,General Medicine,Histology,Pathology and Forensic Medicine

Reference66 articles.

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3. Nemaline myopathy with minicores caused by mutation of the CFL2 gene encoding the skeletal muscle actin-binding protein-cofilin-2;Agrawal;Am. J. Hum. Genet.,2007

4. Normal myofibrillar development followed by progressive sarcomeric disruption with actin accumulations in a mouse Cfl2 knockout demonstrates requirement of cofilin-2 for muscle maintenance;Agrawal;Hum. Mol. Genet.,2012

5. ADF/cofilin and actin dynamics in disease;Bamburg;Trends Cell Biol.,2002

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