Normal myofibrillar development followed by progressive sarcomeric disruption with actin accumulations in a mouse Cfl2 knockout demonstrates requirement of cofilin-2 for muscle maintenance-Reference-Cited by-同舟云学术

Normal myofibrillar development followed by progressive sarcomeric disruption with actin accumulations in a mouse Cfl2 knockout demonstrates requirement of cofilin-2 for muscle maintenance

Published:2012-02-17 Issue:10 Volume:21 Page:2341-2356
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:Human Molecular Genetics

Author:

Agrawal P. B.,Joshi M.,Savic T.,Chen Z.,Beggs A. H.

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference37 articles.

1. Proteins of the ADF/Cofilin Family: Essential Regulators of Actin Dynamics

2. Nemaline Myopathy with Minicores Caused by Mutation of the CFL2 Gene Encoding the Skeletal Muscle Actin–Binding Protein, Cofilin-2

3. Characterization of human muscle type cofilin (CFL2) in normal and regenerating muscle

4. The Three Mouse Actin-depolymerizing Factor/Cofilins Evolved to Fulfill Cell-Type–specific Requirements for Actin Dynamics

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