Chronic Phenotype Characterization of a Large-Animal Model of Hereditary Tyrosinemia Type 1-Reference-Cited by-同舟云学术

Chronic Phenotype Characterization of a Large-Animal Model of Hereditary Tyrosinemia Type 1

Published:2017-01 Issue:1 Volume:187 Page:33-41
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Elgilani Faysal,Mao Shennen A.,Glorioso Jaime M.,Yin Meng,Iankov Ianko D.,Singh Anisha,Amiot Bruce,Rinaldo Piero,Marler Ronald J.,Ehman Richard L.,Grompe Markus,Lillegard Joseph B.,Hickey Raymond D.,Nyberg Scott L.

Funder

NIH

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference33 articles.

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2. “Oxidative stress” response in liver of an untreated newborn mouse having a 1.2-centimorgan deletion on chromosome 7;Liang;Biochem Biophys Res Commun,1992

3. The mutagenicity of the tyrosine metabolite, fumarylacetoacetate, is enhanced by glutathione depletion;Jorquera;Biochem Biophys Res Commun,1997

4. In vivo suppressor mutations correct a murine model of hereditary tyrosinemia type I;Manning;Proc Natl Acad Sci U S A,1999

5. Treatment of hereditary tyrosinaemia type I by inhibition of 4-hydroxyphenylpyruvate dioxygenase;Lindstedt;Lancet,1992

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