Heat Shock Protein 72 Enhances Autophagy as a Protective Mechanism in Lipopolysaccharide-Induced Peritonitis in Rats-Reference-Cited by-同舟云学术

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Heat Shock Protein 72 Enhances Autophagy as a Protective Mechanism in Lipopolysaccharide-Induced Peritonitis in Rats

Published:2011-12 Issue:6 Volume:179 Page:2822-2834
ISSN:0002-9440
Container-title:The American Journal of Pathology
language:en
Short-container-title:The American Journal of Pathology

Author:

Li Shu,Zhou Yi,Fan Jinjin,Cao Shirong,Cao Tao,Huang Fengxian,Zhuang Shougang,Wang Yihan,Yu Xueqing,Mao Haiping

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Publisher

Elsevier BV

Subject

Pathology and Forensic Medicine

Reference64 articles.

1. Intratracheal instillation of lipopolysaccharide in mice induces apoptosis in bronchial epithelial cells: no role for tumor necrosis factor-alpha and infiltrating neutrophils;Vernooy;Am J Respir Cell Mol Biol,2001

2. DNA fragmentation in mouse organs during endotoxic shock;Bohlinger;Am J Pathol,1996

3. Overexpression of Hsp20 prevents endotoxin-induced myocardial dysfunction and apoptosis via inhibition of NF-kappaB activation;Wang;J Mol Cell Cardiol,2009

4. Endothelial cell apoptosis in lipopolysaccharide-induced lung injury in mice;Fujita;Int Arch Allergy Immunol,1998

5. Lipopolysaccharide induces disseminated endothelial apoptosis requiring ceramide generation;Haimovitz-Friedman;J Exp Med,1997

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