Molecular and Functional Key Features and Oncogenic Drivers in Thymic Carcinomas

Author:

Barachini Serena1ORCID,Pardini Eleonora2ORCID,Burzi Irene Sofia2ORCID,Sardo Infirri Gisella2,Montali Marina1ORCID,Petrini Iacopo2ORCID

Affiliation:

1. Department of Translational Research and of New Surgical and Medical Technologies, University of Pisa, 56126 Pisa, Italy

2. Department of Clinical and Experimental Medicine, University of Pisa, 56126 Pisa, Italy

Abstract

Thymic epithelial tumors, comprising thymic carcinomas and thymomas, are rare neoplasms. They differ in histology, prognosis, and association with autoimmune diseases such as myasthenia gravis. Thymomas, but not thymic carcinomas, often harbor GTF2I mutations. Mutations of CDKN2A, TP53, and CDKN2B are the most common thymic carcinomas. The acquisition of mutations in genes that control chromatin modifications and epigenetic regulation occurs in the advanced stages of thymic carcinomas. Anti-angiogenic drugs and immune checkpoint inhibitors targeting the PD-1/PD-L1 axis have shown promising results for the treatment of unresectable tumors. Since thymic carcinomas are frankly aggressive tumors, this report presents insights into their oncogenic drivers, categorized under the established hallmarks of cancer.

Funder

Ail Pisa

Publisher

MDPI AG

Subject

Cancer Research,Oncology

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